Online ISSN: 1945-4589
www.aging-us.com
About Aging
Launched in 2009, Aging publishes papers of general interest and biological significance in all fields of aging research and age-related diseases, including cancer—and now, with a special focus on COVID-19 vulnerability as an age-dependent syndrome. Topics in Aging go beyond traditional gerontology, including, but not limited to, cellular and molecular biology, human age-related diseases, pathology in model organisms, signal transduction pathways (e.g., p53, sirtuins, and PI-3K/AKT/mTOR, among others), and approaches to modulating these signaling pathways.

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Aging | Lamin A to Z in normal aging

Aging

November 9, 2022
PRESS RELEASE: A new research perspective was published in Aging's Volume 14, Issue 20, entitled, “Lamin A to Z in normal aging.” continue reading »

Aging’s Scientific Integrity Process

Aging

November 8, 2022
The open-access journal Aging recently launched a new webpage showcasing the full Aging Scientific Integrity Process. continue reading »

Aging | Rapamycin treatment early in life reprograms aging: hyperfunction theory and clinical practice

Aging

November 3, 2022
PRESS RELEASE: A new research perspective was published on October 24, 2022, in Aging's Volume 14, Issue 20, entitled, “Rapamycin treatment early in life reprograms aging: hyperfunction theory and clinical practice.” continue reading »

Behind the Study: Novel TRKB Agonists Activate TRKB and Downstream ERK and AKT Signaling

Aging

November 1, 2022
Dr. Ya-Jen Chiu discusses a research paper she co-authored that was published by Aging (Aging-US), entitled, “Novel TRKB agonists activate TRKB and downstream ERK and AKT signaling to protect Aβ-GFP SH-SY5Y cells against Aβ toxicity.” continue reading »

Aging | Downregulation of senescence-associated secretory phenotype by knockdown of secreted frizzled-related protein 4 contributes to the prevention of skin aging

Aging

October 31, 2022
PRESS RELEASE: A new research paper was published on the cover of Aging's Volume 14, Issue 20, entitled, “Downregulation of senescence-associated secretory phenotype by knockdown of secreted frizzled-related protein 4 contributes to the prevention of skin aging.” continue reading »