Online ISSN: 1945-4589
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About Aging
Launched in 2009, Aging publishes papers of general interest and biological significance in all fields of aging research and age-related diseases, including cancer—and now, with a special focus on COVID-19 vulnerability as an age-dependent syndrome. Topics in Aging go beyond traditional gerontology, including, but not limited to, cellular and molecular biology, human age-related diseases, pathology in model organisms, signal transduction pathways (e.g., p53, sirtuins, and PI-3K/AKT/mTOR, among others), and approaches to modulating these signaling pathways.

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A Call for Standardization of Controls in Lifespan Studies

Aging

March 5, 2024
PRESS RELEASE: A new research perspective was published in Aging’s Volume 16, Issue 4, entitled, “On standardization of controls in lifespan studies.” continue reading »

Mapping the Core Senescence Phenotype of Human Colon Fibroblasts

Aging

February 29, 2024
PRESS RELEASE:A new research paper was published on the cover of Aging's Volume 16, Issue 4, entitled, “Mapping the core senescence phenotype of primary human colon fibroblasts.” continue reading »

Prognostic Model Development and Molecular Subtypes Identification in Bladder Urothelial Cancer

News

February 28, 2024
PRESS RELEASE: A new research paper was published in Aging's Volume 16, Issue 3, entitled, “Prognostic model development and molecular subtypes identification in bladder urothelial cancer by oxidative stress signatures.” continue reading »

The Anti-aging Effect of Vitamin D and its Receptor in Drosophila Midgut

Aging

February 27, 2024
PRESS RELEASE: A new research paper was published in Aging’s Volume 16, Issue 3, entitled, “The anti-aging effect of vitamin D and vitamin D receptor in Drosophila midgut.” continue reading »

Disruption of Mitochondrial Unfolded Protein Response Results in Shortening of Telomeres in Mouse Oocytes and Somatic Cells

News

February 21, 2024
PRESS RELEASE: A new research paper was published in Aging's Volume 16, Issue 3, entitled, “Disruption of mitochondrial unfolded protein response results in telomere shortening in mouse oocytes and somatic cells.” continue reading »